1. Warmke, Laura M, Wang, Wei-Lien, Baumhoer, Daniel, Baker, Michael L, Kerr, Darcy A. 2023. TAF15::NR4A3 gene fusion identifies a morphologically distinct subset of extraskeletal myxoid chondrosarcoma mimicking myoepithelial tumors. In Genes, chromosomes & cancer, 62, 581-588. doi:10.1002/gcc.23144. https://pubmed.ncbi.nlm.nih.gov/37057757/
2. Jobert, Laure, Argentini, Manuela, Tora, László. 2008. PRMT1 mediated methylation of TAF15 is required for its positive gene regulatory function. In Experimental cell research, 315, 1273-86. doi:10.1016/j.yexcr.2008.12.008. https://pubmed.ncbi.nlm.nih.gov/19124016/
3. Blechingberg, Jenny, Luo, Yonglun, Bolund, Lars, Damgaard, Christian Kroun, Nielsen, Anders Lade. 2012. Gene expression responses to FUS, EWS, and TAF15 reduction and stress granule sequestration analyses identifies FET-protein non-redundant functions. In PloS one, 7, e46251. doi:10.1371/journal.pone.0046251. https://pubmed.ncbi.nlm.nih.gov/23049996/
4. Ticozzi, N, Vance, C, Leclerc, A L, Brown, R H, Landers, J E. 2011. Mutational analysis reveals the FUS homolog TAF15 as a candidate gene for familial amyotrophic lateral sclerosis. In American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics, 156B, 285-90. doi:10.1002/ajmg.b.31158. https://pubmed.ncbi.nlm.nih.gov/21438137/
5. Yao, Li, Cen, Jiannong, Pan, Jinlan, Ruan, Changgeng, Chen, Suning. 2017. TAF15-ZNF384 fusion gene in childhood mixed phenotype acute leukemia. In Cancer genetics, 211, 1-4. doi:10.1016/j.cancergen.2016.12.002. https://pubmed.ncbi.nlm.nih.gov/28279306/
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