1. Goodarzi, Hani, Nguyen, Hoang C B, Zhang, Steven, Molina, Henrik, Tavazoie, Sohail F. . Modulated Expression of Specific tRNAs Drives Gene Expression and Cancer Progression. In Cell, 165, 1416-1427. doi:10.1016/j.cell.2016.05.046. https://pubmed.ncbi.nlm.nih.gov/27259150/
2. Moll, Tobias, Odon, Valerie, Harvey, Calum, Rehwinkel, Jan, Cooper-Knock, Johnathan. 2022. Low expression of EXOSC2 protects against clinical COVID-19 and impedes SARS-CoV-2 replication. In Life science alliance, 6, . doi:10.26508/lsa.202201449. https://pubmed.ncbi.nlm.nih.gov/36241425/
3. Moll, Tobias, Odon, Valerie, Harvey, Calum, Rehwinkel, Jan, Cooper-Knock, Johnathan. 2022. Low expression of EXOSC2 protects against clinical COVID-19 and impedes SARS-CoV-2 replication. In bioRxiv : the preprint server for biology, , . doi:10.1101/2022.03.06.483172. https://pubmed.ncbi.nlm.nih.gov/35291294/
4. Yatsuka, Hiroyuki, Hada, Kazumasa, Shiraishi, Hiroshi, Kubota, Toshiaki, Hanada, Toshikatsu. 2020. Exosc2 deficiency leads to developmental disorders by causing a nucleotide pool imbalance in zebrafish. In Biochemical and biophysical research communications, 533, 1470-1476. doi:10.1016/j.bbrc.2020.10.044. https://pubmed.ncbi.nlm.nih.gov/33333712/
5. Di Donato, Nataliya, Neuhann, Teresa, Kahlert, Anne-Karin, Bier, Andrea, Rump, Andreas. 2016. Mutations in EXOSC2 are associated with a novel syndrome characterised by retinitis pigmentosa, progressive hearing loss, premature ageing, short stature, mild intellectual disability and distinctive gestalt. In Journal of medical genetics, 53, 419-25. doi:10.1136/jmedgenet-2015-103511. https://pubmed.ncbi.nlm.nih.gov/26843489/
6. Yang, Xue, Bayat, Vafa, DiDonato, Nataliya, Khavari, Paul, Lu, Bingwei. . Genetic and genomic studies of pathogenic EXOSC2 mutations in the newly described disease SHRF implicate the autophagy pathway in disease pathogenesis. In Human molecular genetics, 29, 541-553. doi:10.1093/hmg/ddz251. https://pubmed.ncbi.nlm.nih.gov/31628467/
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