1. Jimenez-Vazquez, Eric N, Arad, Michael, Macías, Álvaro, Michele, Daniel E, Jalife, José. 2022. SNTA1 gene rescues ion channel function and is antiarrhythmic in cardiomyocytes derived from induced pluripotent stem cells from muscular dystrophy patients. In eLife, 11, . doi:10.7554/eLife.76576. https://pubmed.ncbi.nlm.nih.gov/35762211/
2. Simon, Matthew, Wang, Marie Xun, Ismail, Ozama, Boison, Detlev, Iliff, Jeffrey J. 2022. Loss of perivascular aquaporin-4 localization impairs glymphatic exchange and promotes amyloid β plaque formation in mice. In Alzheimer's research & therapy, 14, 59. doi:10.1186/s13195-022-00999-5. https://pubmed.ncbi.nlm.nih.gov/35473943/
3. Mestre, Humberto, Hablitz, Lauren M, Xavier, Anna Lr, Iliff, Jeffrey J, Nedergaard, Maiken. 2018. Aquaporin-4-dependent glymphatic solute transport in the rodent brain. In eLife, 7, . doi:10.7554/eLife.40070. https://pubmed.ncbi.nlm.nih.gov/30561329/
4. Lieve, Krystien V, Williams, Leah, Daly, Amy, Macaya, Daniela, Chung, Wendy K. 2013. Results of genetic testing in 855 consecutive unrelated patients referred for long QT syndrome in a clinical laboratory. In Genetic testing and molecular biomarkers, 17, 553-61. doi:10.1089/gtmb.2012.0118. https://pubmed.ncbi.nlm.nih.gov/23631430/
5. Tan, Xiaoli, Li, Xiaohong, Li, Ruhua, Li, Li, Li, Hao. 2024. β-hydroxybutyrate alleviates neurological deficits by restoring glymphatic and inflammation after subarachnoid hemorrhage in mice. In Experimental neurology, 378, 114819. doi:10.1016/j.expneurol.2024.114819. https://pubmed.ncbi.nlm.nih.gov/38763355/
6. Wu, Geru, Ai, Tomohiko, Kim, Jeffrey J, Cheng, Jie, Vatta, Matteo. . alpha-1-syntrophin mutation and the long-QT syndrome: a disease of sodium channel disruption. In Circulation. Arrhythmia and electrophysiology, 1, 193-201. doi:10.1161/CIRCEP.108.769224. https://pubmed.ncbi.nlm.nih.gov/19684871/
7. Pedersen, Taylor J, Keil, Samantha A, Han, Warren, Wang, Marie X, Iliff, Jeffrey J. 2023. The effect of aquaporin-4 mis-localization on Aβ deposition in mice. In Neurobiology of disease, 181, 106100. doi:10.1016/j.nbd.2023.106100. https://pubmed.ncbi.nlm.nih.gov/36990365/