1. Colgan, Diana F, Goodfellow, Renee X, Colgan, John D. 2021. The transcriptional regulator GON4L is required for viability and hematopoiesis in mice. In Experimental hematology, 98, 25-35. doi:10.1016/j.exphem.2021.04.001. https://pubmed.ncbi.nlm.nih.gov/33864850/
2. Budine, Terin E, de Sena-Tomás, Carmen, Williams, Margot L K, Targoff, Kimara L, Solnica-Krezel, Lila. 2020. Gon4l/Udu regulates cardiomyocyte proliferation and maintenance of ventricular chamber identity during zebrafish development. In Developmental biology, 462, 223-234. doi:10.1016/j.ydbio.2020.03.002. https://pubmed.ncbi.nlm.nih.gov/32272116/
3. Lu, Ping, Hankel, Isaiah L, Knisz, Judit, Rothman, Paul B, Colgan, John D. 2010. The Justy mutation identifies Gon4-like as a gene that is essential for B lymphopoiesis. In The Journal of experimental medicine, 207, 1359-67. doi:10.1084/jem.20100147. https://pubmed.ncbi.nlm.nih.gov/20530203/
4. Agarwal, Neeraj, Dancik, Garrett M, Goodspeed, Andrew, Duex, Jason E, Theodorescu, Dan. 2016. GON4L Drives Cancer Growth through a YY1-Androgen Receptor-CD24 Axis. In Cancer research, 76, 5175-85. doi:10.1158/0008-5472.CAN-16-1099. https://pubmed.ncbi.nlm.nih.gov/27312530/
5. Barr, Jennifer Y, Goodfellow, Renee X, Colgan, Diana F, Colgan, John D. 2017. Early B Cell Progenitors Deficient for GON4L Fail To Differentiate Due to a Block in Mitotic Cell Division. In Journal of immunology (Baltimore, Md. : 1950), 198, 3978-3988. doi:10.4049/jimmunol.1602054. https://pubmed.ncbi.nlm.nih.gov/28381640/