1. Wu, Kevin Z L, Jones, Rebecca A, Tachie-Menson, Theresa, Smith, James C, Sapkota, Gopal P. 2019. Pathogenic FAM83G palmoplantar keratoderma mutations inhibit the PAWS1:CK1α association and attenuate Wnt signalling. In Wellcome open research, 4, 133. doi:10.12688/wellcomeopenres.15403.2. https://pubmed.ncbi.nlm.nih.gov/31656861/
2. Liu, Chen, Huang, Xuan-Yu, Huang, Yun. 2021. FAM83G promotes proliferation, invasion, and metastasis by regulating PI3K/AKT signaling in hepatocellular carcinoma cells. In Biochemical and biophysical research communications, 567, 63-71. doi:10.1016/j.bbrc.2021.05.081. https://pubmed.ncbi.nlm.nih.gov/34144502/
3. Jin, Yi, Yu, Jiahui, Jiang, Yi, Gu, Xi, Zhu, Xudong. 2022. Comprehensive analysis of the expression, prognostic significance, and function of FAM83 family members in breast cancer. In World journal of surgical oncology, 20, 172. doi:10.1186/s12957-022-02636-9. https://pubmed.ncbi.nlm.nih.gov/35650627/
4. Glennie, Lorraine, Solà, Marta Codina, Xunclà, Mar, Lasa-Aranzasti, Amaia, Sapkota, Gopal P. 2024. A novel FAM83G variant from palmoplantar keratoderma patient disrupts WNT signalling via loss of FAM83G-CK1α interaction. In Open biology, 14, 240075. doi:10.1098/rsob.240075. https://pubmed.ncbi.nlm.nih.gov/39043225/
5. Ikeda, Takako, Honda, Chikako Kanno, Kurozumi, Sasagu, Shirabe, Ken, Fujii, Takaaki. . Clinical Significance of FAM83G in Breast Cancer: Association With Triple-negative Subtype and Prognosis. In Anticancer research, 44, 4877-4883. doi:10.21873/anticanres.17313. https://pubmed.ncbi.nlm.nih.gov/39477305/
6. Vogt, Janis, Dingwell, Kevin S, Herhaus, Lina, Smith, James C, Sapkota, Gopal P. 2014. Protein associated with SMAD1 (PAWS1/FAM83G) is a substrate for type I bone morphogenetic protein receptors and modulates bone morphogenetic protein signalling. In Open biology, 4, 130210. doi:10.1098/rsob.130210. https://pubmed.ncbi.nlm.nih.gov/24554596/
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