1. Luo, Haiying, Hu, Xiaoxia, Li, Yadi, Zeng, Yueying, Qin, Bo. 2022. The antiviral activity of tripartite motif protein 38 in hepatitis B virus replication and gene expression and its association with treatment responses during PEG-IFN-α antiviral therapy. In Virology, 579, 84-93. doi:10.1016/j.virol.2022.12.014. https://pubmed.ncbi.nlm.nih.gov/36623352/
2. Kim, Kabsun, Kim, Jung Ha, Kim, Inyoung, Seong, Semun, Kim, Nacksung. 2018. TRIM38 regulates NF-κB activation through TAB2 degradation in osteoclast and osteoblast differentiation. In Bone, 113, 17-28. doi:10.1016/j.bone.2018.05.009. https://pubmed.ncbi.nlm.nih.gov/29753717/
3. Sun, Qingqing, Han, Xiao, Meng, Lingtong, Wei, Lin, Ma, Cuiqing. 2024. TRIM38 Induced in Respiratory Syncytial Virus-infected Cells Downregulates Type I Interferon Expression by Competing with TRIM25 to Bind RIG-I. In Inflammation, 47, 1328-1343. doi:10.1007/s10753-024-01979-7. https://pubmed.ncbi.nlm.nih.gov/38630167/
4. Wang, Xiaojing, He, Hongchao, Rui, Wenbin, Zhu, Yu, Xie, Xin. 2021. TRIM38 triggers the uniquitination and degradation of glucose transporter type 1 (GLUT1) to restrict tumor progression in bladder cancer. In Journal of translational medicine, 19, 508. doi:10.1186/s12967-021-03173-x. https://pubmed.ncbi.nlm.nih.gov/34906161/
5. Hu, Ming-Ming, Xie, Xue-Qin, Yang, Qing, Lin, Heng, Shu, Hong-Bing. 2015. TRIM38 Negatively Regulates TLR3/4-Mediated Innate Immune and Inflammatory Responses by Two Sequential and Distinct Mechanisms. In Journal of immunology (Baltimore, Md. : 1950), 195, 4415-25. doi:10.4049/jimmunol.1500859. https://pubmed.ncbi.nlm.nih.gov/26392463/
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