1. Zheng, Li, Pan, Cong, Tian, Wanli, Zhu, Xianjun, Hu, Yiguo. 2023. Atp8a1 deletion increases the proliferative activity of hematopoietic stem cells by impairing PTEN function. In Cellular oncology (Dordrecht, Netherlands), 46, 1069-1083. doi:10.1007/s13402-023-00797-7. https://pubmed.ncbi.nlm.nih.gov/36930333/
2. Yap, Yi T, Li, Yu H, Li, Wei, Banerjee, Probal, Zhang, Zhibing. 2021. ATP8a1, an IFT27 binding partner, is dispensable for spermatogenesis and male fertility. In Molecular reproduction and development, 88, 371-375. doi:10.1002/mrd.23470. https://pubmed.ncbi.nlm.nih.gov/33821543/
3. Jing, Weidong, Yabas, Mehmet, Bröer, Angelika, Enders, Anselm, Bröer, Stefan. . Calpain cleaves phospholipid flippase ATP8A1 during apoptosis in platelets. In Blood advances, 3, 219-229. doi:10.1182/bloodadvances.2018023473. https://pubmed.ncbi.nlm.nih.gov/30674456/
4. Kook, Seunghyi, Wang, Ping, Meng, Shufang, Marks, Michael S, Guttentag, Susan H. . AP-3-dependent targeting of flippase ATP8A1 to lamellar bodies suppresses activation of YAP in alveolar epithelial type 2 cells. In Proceedings of the National Academy of Sciences of the United States of America, 118, . doi:10.1073/pnas.2025208118. https://pubmed.ncbi.nlm.nih.gov/33990468/
5. Pocognoni, Cristian A, Nawara, Tomasz, Bhatt, Jay M, Randazzo, Paul, Sztul, Elizabeth. 2024. The lipid flippase ATP8A1 regulates the recruitment of ARF effectors to the trans-Golgi Network. In Archives of biochemistry and biophysics, 758, 110049. doi:10.1016/j.abb.2024.110049. https://pubmed.ncbi.nlm.nih.gov/38879142/
6. Levano, Kelly, Punia, Vineet, Raghunath, Michael, Fata, Jimmie, Banerjee, Probal. 2011. Atp8a1 deficiency is associated with phosphatidylserine externalization in hippocampus and delayed hippocampus-dependent learning. In Journal of neurochemistry, 120, 302-13. doi:10.1111/j.1471-4159.2011.07543.x. https://pubmed.ncbi.nlm.nih.gov/22007859/
7. Kerr, Daniel J, Marsillo, Alexandra, Guariglia, Sara R, Wieraszko, Andrzej, Banerjee, Probal. 2016. Aberrant hippocampal Atp8a1 levels are associated with altered synaptic strength, electrical activity, and autistic-like behavior. In Biochimica et biophysica acta, 1862, 1755-65. doi:10.1016/j.bbadis.2016.06.005. https://pubmed.ncbi.nlm.nih.gov/27287255/
智鼠故事 
