1. Zheng, Danni, Fu, Weida, Jin, Lingli, Guan, Yaoyao, Hao, Rutian. 2022. The Overexpression and Clinical Significance of AP1S1 in Breast Cancer. In Cancer management and research, 14, 1475-1492. doi:10.2147/CMAR.S346519. https://pubmed.ncbi.nlm.nih.gov/35463798/
2. Jeong, Jangho, Hwang, Ye Eun, Lee, Minwoo, Choi, Jee-Hye, Rhee, Sangmyung. 2023. Downregulation of AP1S1 causes the lysosomal degradation of EGFR in non-small cell lung cancer. In Journal of cellular physiology, 238, 2335-2347. doi:10.1002/jcp.31112. https://pubmed.ncbi.nlm.nih.gov/37659097/
3. Klee, Katharina M C, Janecke, Andreas R, Civan, Hasret A, Müller, Thomas, Vogel, Georg F. 2020. AP1S1 missense mutations cause a congenital enteropathy via an epithelial barrier defect. In Human genetics, 139, 1247-1259. doi:10.1007/s00439-020-02168-w. https://pubmed.ncbi.nlm.nih.gov/32306098/
4. Martinelli, Diego, Dionisi-Vici, Carlo. 2014. AP1S1 defect causing MEDNIK syndrome: a new adaptinopathy associated with defective copper metabolism. In Annals of the New York Academy of Sciences, 1314, 55-63. doi:10.1111/nyas.12426. https://pubmed.ncbi.nlm.nih.gov/24754424/
5. Su, Feng, Fang, Yong, Yu, Jinjie, Tan, Lijie, Yin, Jun. 2022. The Single Nucleotide Polymorphisms of AP1S1 are Associated with Risk of Esophageal Squamous Cell Carcinoma in Chinese Population. In Pharmacogenomics and personalized medicine, 15, 235-247. doi:10.2147/PGPM.S342743. https://pubmed.ncbi.nlm.nih.gov/35321090/