1. Deng, Tongyuan, Shen, Peng, Li, Aimin, Liu, Zhen, Fang, Weiyi. 2021. CCDC65 as a new potential tumor suppressor induced by metformin inhibits activation of AKT1 via ubiquitination of ENO1 in gastric cancer. In Theranostics, 11, 8112-8128. doi:10.7150/thno.54961. https://pubmed.ncbi.nlm.nih.gov/34335983/
2. Zhang, Ziyan, Xu, Ping, Hu, Zhe, Li, Rong, Fang, Weiyi. 2022. CCDC65, a Gene Knockout that leads to Early Death of Mice, acts as a potentially Novel Tumor Suppressor in Lung Adenocarcinoma. In International journal of biological sciences, 18, 4171-4186. doi:10.7150/ijbs.69332. https://pubmed.ncbi.nlm.nih.gov/35844805/
3. Horani, Amjad, Brody, Steven L, Ferkol, Thomas W, Elpeleg, Orly, Kerem, Eitan. 2013. CCDC65 mutation causes primary ciliary dyskinesia with normal ultrastructure and hyperkinetic cilia. In PloS one, 8, e72299. doi:10.1371/journal.pone.0072299. https://pubmed.ncbi.nlm.nih.gov/23991085/
4. Ben Braiek, Maxime, Moreno-Romieux, Carole, Allain, Charlotte, Woloszyn, Florent, Fabre, Stéphane. 2021. A Nonsense Variant in CCDC65 Gene Causes Respiratory Failure Associated with Increased Lamb Mortality in French Lacaune Dairy Sheep. In Genes, 13, . doi:10.3390/genes13010045. https://pubmed.ncbi.nlm.nih.gov/35052387/
5. Bower, Raqual, Tritschler, Douglas, Mills, Kristyn VanderWaal, Nicastro, Daniela, Porter, Mary E. 2017. DRC2/CCDC65 is a central hub for assembly of the nexin-dynein regulatory complex and other regulators of ciliary and flagellar motility. In Molecular biology of the cell, 29, 137-153. doi:10.1091/mbc.E17-08-0510. https://pubmed.ncbi.nlm.nih.gov/29167384/
6. Austin-Tse, Christina, Halbritter, Jan, Zariwala, Maimoona A, Drummond, Iain A, Hildebrandt, Friedhelm. . Zebrafish Ciliopathy Screen Plus Human Mutational Analysis Identifies C21orf59 and CCDC65 Defects as Causing Primary Ciliary Dyskinesia. In American journal of human genetics, 93, 672-86. doi:10.1016/j.ajhg.2013.08.015. https://pubmed.ncbi.nlm.nih.gov/24094744/
智鼠故事 
