1. Yamada, Shuhei. 2020. Specific functions of Exostosin-like 3 (EXTL3) gene products. In Cellular & molecular biology letters, 25, 39. doi:10.1186/s11658-020-00231-y. https://pubmed.ncbi.nlm.nih.gov/32843889/
2. Bajaj, Shruti, Satoskar, Purnima, Nair, Aadhira, Sheth, Jayesh, Sheth, Harsh. 2022. An ultra-rare case of immunoskeletal dysplasia with neurodevelopmental abnormalities in an Indian patient with homozygous c.953C > T variant in EXTL3 gene: a case report. In BMC pediatrics, 22, 78. doi:10.1186/s12887-022-03143-2. https://pubmed.ncbi.nlm.nih.gov/35114981/
3. Kaidonis, Xenia, Liaw, Wan Chin, Roberts, Ainslie Derrick, Anson, Donald, Byers, Sharon. 2009. Gene silencing of EXTL2 and EXTL3 as a substrate deprivation therapy for heparan sulphate storing mucopolysaccharidoses. In European journal of human genetics : EJHG, 18, 194-9. doi:10.1038/ejhg.2009.143. https://pubmed.ncbi.nlm.nih.gov/19690583/
4. Chang, Pingan, Chen, Shenglan, Chang, Xiumei, Tang, Qingsheng, Ma, Limin. 2022. EXTL3 could serve as a potential biomarker of prognosis and immunotherapy for prostate cancer and its potential mechanisms. In European journal of medical research, 27, 115. doi:10.1186/s40001-022-00740-w. https://pubmed.ncbi.nlm.nih.gov/35818069/
5. Huang, Yuege, Mei, Hong, Deng, Chunchen, Li, Jia-Da, Liu, Jia. 2024. EXTL3 and NPC1 are mammalian host factors for Autographa californica multiple nucleopolyhedrovirus infection. In Nature communications, 15, 7711. doi:10.1038/s41467-024-52193-w. https://pubmed.ncbi.nlm.nih.gov/39231976/
6. Karibe, T, Fukui, H, Sekikawa, A, Shiratori, K, Fujimori, T. . EXTL3 promoter methylation down-regulates EXTL3 and heparan sulphate expression in mucinous colorectal cancers. In The Journal of pathology, 216, 32-42. doi:10.1002/path.2377. https://pubmed.ncbi.nlm.nih.gov/18543267/
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