1. Liu, Zhanju, Liu, Ruize, Gao, Han, Li, Mingsong, Huang, Hailiang. 2023. Genetic architecture of the inflammatory bowel diseases across East Asian and European ancestries. In Nature genetics, 55, 796-806. doi:10.1038/s41588-023-01384-0. https://pubmed.ncbi.nlm.nih.gov/37156999/
2. Le, Huijuan, Wang, Yanyan, Zhou, Jiefei, Cai, Wei, Wu, Jin. 2024. Git2 deficiency promotes MDSCs recruitment in intestine via NF-κB-CXCL1/CXCL12 pathway and ameliorates necrotizing enterocolitis. In Mucosal immunology, 17, 1060-1071. doi:10.1016/j.mucimm.2024.07.006. https://pubmed.ncbi.nlm.nih.gov/39074614/
3. Lu, Daoyuan, Cai, Huan, Park, Sung-Soo, Martin, Bronwen, Maudsley, Stuart. 2015. Nuclear GIT2 is an ATM substrate and promotes DNA repair. In Molecular and cellular biology, 35, 1081-96. doi:10.1128/MCB.01432-14. https://pubmed.ncbi.nlm.nih.gov/25605334/
4. Zobdeh, Farzin, Eremenko, Ivan I, Akan, Mikail A, Schiöth, Helgi B, Mwinyi, Jessica. 2023. The Epigenetics of Migraine. In International journal of molecular sciences, 24, . doi:10.3390/ijms24119127. https://pubmed.ncbi.nlm.nih.gov/37298078/
5. Li, Hui, Guan, Shi-Bing, Lu, Yi, Liu, Yu-Hong, Liu, Qi-Yun. 2017. Genetic deletion of GIT2 prolongs functional recovery and suppresses chondrocyte differentiation in rats with rheumatoid arthritis. In Journal of cellular biochemistry, 119, 1538-1547. doi:10.1002/jcb.26313. https://pubmed.ncbi.nlm.nih.gov/28777475/
6. Lim, Ratana, Lappas, Martha. . GIT2 deficiency attenuates inflammation-induced expression of pro-labor mediators in human amnion and myometrial cells†. In Biology of reproduction, 100, 1617-1629. doi:10.1093/biolre/ioz041. https://pubmed.ncbi.nlm.nih.gov/30915469/
7. Wang, Yanna, Wang, Changyuan, Gong, Ying, Liu, Mozhen, Sun, Huijun. 2025. GIT2 negatively regulates the NF-κB pathway directly or indirectly by regulating TRAF3 expression to promote osteogenic differentiation of BMSCs. In Tissue & cell, 94, 102790. doi:10.1016/j.tice.2025.102790. https://pubmed.ncbi.nlm.nih.gov/39954559/