1. Vall-Palomar, Mònica, Burballa, Carla, Claverie-Martín, Félix, Meseguer, Anna, Ariceta, Gema. 2021. Heterogeneity is a common ground in familial hypomagnesemia with hypercalciuria and nephrocalcinosis caused by CLDN19 gene mutations. In Journal of nephrology, 34, 2053-2062. doi:10.1007/s40620-021-01054-6. https://pubmed.ncbi.nlm.nih.gov/33929692/
2. Yamaguti, Paulo Marcio, Neves, Francisco de Assis Rocha, Hotton, Dominique, Berdal, Ariane, Acevedo, Ana Carolina. 2016. Amelogenesis imperfecta in familial hypomagnesaemia and hypercalciuria with nephrocalcinosis caused by CLDN19 gene mutations. In Journal of medical genetics, 54, 26-37. doi:10.1136/jmedgenet-2016-103956. https://pubmed.ncbi.nlm.nih.gov/27530400/
3. Dimke, Henrik, Griveau, Camille, Ling, Wung-Man Evelyne, Houillier, Pascal, Prot-Bertoye, Caroline. 2023. Claudin-19 localizes to the thick ascending limb where its expression is required for junctional claudin-16 localization. In Annals of the New York Academy of Sciences, 1526, 126-137. doi:10.1111/nyas.15014. https://pubmed.ncbi.nlm.nih.gov/37344378/
4. Vall-Palomar, Mònica, Madariaga, Leire, Ariceta, Gema. 2021. Familial hypomagnesemia with hypercalciuria and nephrocalcinosis. In Pediatric nephrology (Berlin, Germany), 36, 3045-3055. doi:10.1007/s00467-021-04968-2. https://pubmed.ncbi.nlm.nih.gov/33595712/
5. Konrad, Martin, Schaller, Andre, Seelow, Dominik, Nurnberg, Peter, Weber, Stefanie. 2006. Mutations in the tight-junction gene claudin 19 (CLDN19) are associated with renal magnesium wasting, renal failure, and severe ocular involvement. In American journal of human genetics, 79, 949-57. doi:. https://pubmed.ncbi.nlm.nih.gov/17033971/
6. Brideau, Gaelle, Cheval, Lydie, Griveau, Camille, Houillier, Pascal, Prot-Bertoye, Caroline. 2024. Claudin-10 Expression and the Gene Expression Pattern of Thick Ascending Limb Cells. In International journal of molecular sciences, 25, . doi:10.3390/ijms25074008. https://pubmed.ncbi.nlm.nih.gov/38612818/