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2. Li, Haiwen, Wang, Peipei, Hsu, Ethan, Stanford, Kristin I, Han, Renzhi. 2022. Systemic AAV9.BVES delivery ameliorates muscular dystrophy in a mouse model of LGMDR25. In Molecular therapy : the journal of the American Society of Gene Therapy, 31, 398-408. doi:10.1016/j.ymthe.2022.11.012. https://pubmed.ncbi.nlm.nih.gov/36433649/
3. Shi, Yan, Li, Yongqing, Wang, Yuequn, Wu, Xiushan, Yuan, Wuzhou. 2019. The Functional Polymorphism R129W in the BVES Gene Is Associated with Sporadic Tetralogy of Fallot in the Han Chinese Population. In Genetic testing and molecular biomarkers, 23, 601-609. doi:10.1089/gtmb.2019.0085. https://pubmed.ncbi.nlm.nih.gov/31386585/
4. Wu, Min, Li, Ying, He, Xin, Zhang, Cuifang, Zhou, Lei. 2013. Mutational and functional analysis of the BVES gene coding region in Chinese patients with non-syndromic tetralogy of Fallot. In International journal of molecular medicine, 31, 899-903. doi:10.3892/ijmm.2013.1275. https://pubmed.ncbi.nlm.nih.gov/23403794/
5. Shi, Yan, Li, Yongqing, Wang, Yuequn, Wu, Xiushan, Yuan, Wuzhou. 2020. BVES downregulation in non-syndromic tetralogy of fallot is associated with ventricular outflow tract stenosis. In Scientific reports, 10, 14167. doi:10.1038/s41598-020-70806-4. https://pubmed.ncbi.nlm.nih.gov/32843646/
6. Liu, Jin-Xin, Huang, Tong, Xie, Dawei, Yu, Qihong. 2022. Bves maintains vascular smooth muscle cell contractile phenotype and protects against transplant vasculopathy via Dusp1-dependent p38MAPK and ERK1/2 signaling. In Atherosclerosis, 357, 20-32. doi:10.1016/j.atherosclerosis.2022.08.010. https://pubmed.ncbi.nlm.nih.gov/36037759/
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