1. Wang, Min-Jun, Wang, Yuan-Chen, Masson, Emmanuelle, Zou, Wen-Bin, Liao, Zhuan. 2024. SEC16A Variants Predispose to Chronic Pancreatitis by Impairing ER-to-Golgi Transport and Inducing ER Stress. In Advanced science (Weinheim, Baden-Wurttemberg, Germany), 11, e2402550. doi:10.1002/advs.202402550. https://pubmed.ncbi.nlm.nih.gov/39119875/
2. Piao, He, Kim, Jiyoon, Noh, Shin Hye, Kim, Joo Young, Lee, Min Goo. 2017. Sec16A is critical for both conventional and unconventional secretion of CFTR. In Scientific reports, 7, 39887. doi:10.1038/srep39887. https://pubmed.ncbi.nlm.nih.gov/28067262/
3. Wu, Yachen, Zheng, Wenzhong, Xu, Guofeng, Wang, Lianrong, Chen, Shi. 2024. C9orf72 controls hepatic lipid metabolism by regulating SREBP1 transport. In Cell death and differentiation, 31, 1070-1084. doi:10.1038/s41418-024-01312-7. https://pubmed.ncbi.nlm.nih.gov/38816580/
4. Bruno, Joanne, Brumfield, Alexandria, Chaudhary, Natasha, Iaea, David, McGraw, Timothy E. 2016. SEC16A is a RAB10 effector required for insulin-stimulated GLUT4 trafficking in adipocytes. In The Journal of cell biology, 214, 61-76. doi:10.1083/jcb.201509052. https://pubmed.ncbi.nlm.nih.gov/27354378/
5. Mayer, Louisa, Jasztal, Maria, Pardo, Mercedes, Ouwehand, Willem H, Guerrero, Jose A. 2017. Nbeal2 interacts with Dock7, Sec16a, and Vac14. In Blood, 131, 1000-1011. doi:10.1182/blood-2017-08-800359. https://pubmed.ncbi.nlm.nih.gov/29187380/
6. Hu, Shixian, Vich Vila, Arnau, Gacesa, Ranko, Kurilshikov, Alexander, Weersma, Rinse K. 2020. Whole exome sequencing analyses reveal gene-microbiota interactions in the context of IBD. In Gut, 70, 285-296. doi:10.1136/gutjnl-2019-319706. https://pubmed.ncbi.nlm.nih.gov/32651235/
7. Cho, Hyun Jin, Yu, Jia, Xie, Chengsong, Liu, Zhihua, Cai, Huaibin. 2014. Leucine-rich repeat kinase 2 regulates Sec16A at ER exit sites to allow ER-Golgi export. In The EMBO journal, 33, 2314-31. doi:10.15252/embj.201487807. https://pubmed.ncbi.nlm.nih.gov/25201882/
8. O'Rielly, Darren D, Uddin, Mohammed, Codner, Dianne, Inman, Robert, Rahman, Proton. 2015. Private rare deletions in SEC16A and MAMDC4 may represent novel pathogenic variants in familial axial spondyloarthritis. In Annals of the rheumatic diseases, 75, 772-9. doi:10.1136/annrheumdis-2014-206484. https://pubmed.ncbi.nlm.nih.gov/25956157/
智鼠故事 
