1. Pataskar, Abhijeet, Champagne, Julien, Nagel, Remco, Peeper, Daniel S, Agami, Reuven. 2022. Tryptophan depletion results in tryptophan-to-phenylalanine substitutants. In Nature, 603, 721-727. doi:10.1038/s41586-022-04499-2. https://pubmed.ncbi.nlm.nih.gov/35264796/
2. Megarbane, Andre, Bizzari, Sami, Deepthi, Asha, Delague, Valérie, Urtizberea, J Andoni. . A 20-year Clinical and Genetic Neuromuscular Cohort Analysis in Lebanon: An International Effort. In Journal of neuromuscular diseases, 9, 193-210. doi:10.3233/JND-210652. https://pubmed.ncbi.nlm.nih.gov/34602496/
3. Kim, Yoon Tae, Huh, Jin Won, Choi, Yun Hui, Kim, Sunghoon, Jin, Mirim. 2023. Highly secreted tryptophanyl tRNA synthetase 1 as a potential theranostic target for hypercytokinemic severe sepsis. In EMBO molecular medicine, 16, 40-63. doi:10.1038/s44321-023-00004-y. https://pubmed.ncbi.nlm.nih.gov/38177528/
4. Bögershausen, Nina, Krawczyk, Hannah E, Jamra, Rami A, Kratz, Christian P, Wollnik, Bernd. 2022. WARS1 and SARS1: Two tRNA synthetases implicated in autosomal recessive microcephaly. In Human mutation, 43, 1454-1471. doi:10.1002/humu.24430. https://pubmed.ncbi.nlm.nih.gov/35790048/
5. Oshima, Takashi, Hashimoto, Itaru, Hiroshima, Yukihiko, Saito, Aya, Miyagi, Yohei. . Clinical Significance of Tryptophanyl-tRNA Synthetase 1 Gene Expression in Patients With Locally Advanced Gastric Cancer. In Anticancer research, 44, 673-678. doi:10.21873/anticanres.16857. https://pubmed.ncbi.nlm.nih.gov/38307580/
6. Tokolyi, Alex, Persyn, Elodie, Nath, Artika P, Davenport, Emma E, Paul, Dirk S. 2025. The contribution of genetic determinants of blood gene expression and splicing to molecular phenotypes and health outcomes. In Nature genetics, 57, 616-625. doi:10.1038/s41588-025-02096-3. https://pubmed.ncbi.nlm.nih.gov/40038547/
7. Chuang, Tsai-Der, Quintanilla, Derek, Boos, Drake, Khorram, Omid. 2022. Further characterization of tryptophan metabolism and its dysregulation in fibroids. In F&S science, 3, 392-400. doi:10.1016/j.xfss.2022.04.005. https://pubmed.ncbi.nlm.nih.gov/35715314/
8. Lin, Sheng-Jia, Vona, Barbara, Porter, Hillary M, Kim, Hyung-Goo, Varshney, Gaurav K. 2022. Biallelic variants in WARS1 cause a highly variable neurodevelopmental syndrome and implicate a critical exon for normal auditory function. In Human mutation, 43, 1472-1489. doi:10.1002/humu.24435. https://pubmed.ncbi.nlm.nih.gov/35815345/
智鼠故事 
