1. Xiang, Qian-Ming, Jiang, Ni, Liu, Yue-Feng, Guo, Qiang, Li, Kai. 2023. Overexpression of SH2D1A promotes cancer progression and is associated with immune cell infiltration in hepatocellular carcinoma via bioinformatics and in vitro study. In BMC cancer, 23, 1005. doi:10.1186/s12885-023-11315-1. https://pubmed.ncbi.nlm.nih.gov/37858067/
2. Okamoto, Susumu, Ji, Hongbin, Howie, Duncan, Coyle, Anthony J, Terhorst, Cox. . Expression of the SH2D1A gene is regulated by a combination of transcriptional and post-transcriptional mechanisms. In European journal of immunology, 34, 3176-86. doi:. https://pubmed.ncbi.nlm.nih.gov/15459902/
3. Sylla, B S, Murphy, K, Cahir-McFarland, E, Mosialos, G, Kieff, E. . The X-linked lymphoproliferative syndrome gene product SH2D1A associates with p62dok (Dok1) and activates NF-kappa B. In Proceedings of the National Academy of Sciences of the United States of America, 97, 7470-5. doi:. https://pubmed.ncbi.nlm.nih.gov/10852966/
4. Boggio, Elena, Melensi, Matteo, Bocca, Sara, Ramenghi, Ugo, Dianzani, Umberto. 2012. The -346T polymorphism of the SH2D1A gene is a risk factor for development of autoimmunity/lymphoproliferation in males with defective Fas function. In Human immunology, 73, 585-92. doi:10.1016/j.humimm.2012.02.025. https://pubmed.ncbi.nlm.nih.gov/22425739/