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2. Gurumurthy, Aishwarya, Wu, Qiong, Nar, Rukiye, Qian, Zhijian, Bungert, Jörg. 2020. TFII-I/Gtf2i and Erythro-Megakaryopoiesis. In Frontiers in physiology, 11, 590180. doi:10.3389/fphys.2020.590180. https://pubmed.ncbi.nlm.nih.gov/33101065/
3. Wu, Ziyan, Li, Haolong, Xu, Honglin, Wang, Li, Li, Yongzhe. 2023. ChIP-seq analysis found IL21R, a target gene of GTF2I-the susceptibility gene for primary biliary cholangitis in Chinese Han. In Hepatology international, 18, 509-516. doi:10.1007/s12072-023-10586-x. https://pubmed.ncbi.nlm.nih.gov/37713154/
4. Adams, Jason W, Vinokur, Annabelle, de Souza, Janaína S, Wahlin, Karl J, Muotri, Alysson R. 2024. Loss of GTF2I promotes neuronal apoptosis and synaptic reduction in human cellular models of neurodevelopment. In Cell reports, 43, 113867. doi:10.1016/j.celrep.2024.113867. https://pubmed.ncbi.nlm.nih.gov/38416640/
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6. Sakurai, Takeshi, Dorr, Nathan P, Takahashi, Nagahide, Elder, Gregory A, Buxbaum, Joseph D. 2010. Haploinsufficiency of Gtf2i, a gene deleted in Williams Syndrome, leads to increases in social interactions. In Autism research : official journal of the International Society for Autism Research, 4, 28-39. doi:10.1002/aur.169. https://pubmed.ncbi.nlm.nih.gov/21328569/
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