1. Kuwabara, Yasuhide, York, Allen J, Lin, Suh-Chin, Pirruccello, James P, Molkentin, Jeffery D. 2023. A human FLII gene variant alters sarcomeric actin thin filament length and predisposes to cardiomyopathy. In Proceedings of the National Academy of Sciences of the United States of America, 120, e2213696120. doi:10.1073/pnas.2213696120. https://pubmed.ncbi.nlm.nih.gov/37126682/
2. Ruijmbeek, Claudine Wb, Housley, Filomena, Idrees, Hafiza, Verhagen, Judith Ma, Reischauer, Sven. 2023. Biallelic variants in FLII cause pediatric cardiomyopathy by disrupting cardiomyocyte cell adhesion and myofibril organization. In JCI insight, 8, . doi:10.1172/jci.insight.168247. https://pubmed.ncbi.nlm.nih.gov/37561591/
3. Kopecki, Zlatko, Arkell, Ruth M, Strudwick, Xanthe L, Murrell, Dedee F, Cowin, Allison J. 2011. Overexpression of the Flii gene increases dermal-epidermal blistering in an autoimmune ColVII mouse model of epidermolysis bullosa acquisita. In The Journal of pathology, 225, 401-13. doi:10.1002/path.2973. https://pubmed.ncbi.nlm.nih.gov/21984127/
4. Nguyen, Mai Thi, Ly, Quoc Kiet, Kim, Hyun-Jung, Lee, Wan. 2023. FLII Modulates the Myogenic Differentiation of Progenitor Cells via Actin Remodeling-Mediated YAP1 Regulation. In International journal of molecular sciences, 24, . doi:10.3390/ijms241814335. https://pubmed.ncbi.nlm.nih.gov/37762638/
5. Park, Jung-Eun, Lee, Eun Ji, Kim, Jung Kwan, Choi, Jang Hyun, Kang, Min-Ji. 2018. Flightless-I Controls Fat Storage in Drosophila. In Molecules and cells, 41, 603-611. doi:10.14348/molcells.2018.0120. https://pubmed.ncbi.nlm.nih.gov/29890821/