1. Zuo, Xiaofeng, Lobo, Glenn, Fulmer, Diana, Norris, Russell A, Lipschutz, Joshua H. 2019. The exocyst acting through the primary cilium is necessary for renal ciliogenesis, cystogenesis, and tubulogenesis. In The Journal of biological chemistry, 294, 6710-6718. doi:10.1074/jbc.RA118.006527. https://pubmed.ncbi.nlm.nih.gov/30824539/
2. Ortega, Michael A, Villiger, Ross K, Harrison-Chau, Malia, Kepler, Joshua, Fogelgren, Ben. 2022. Exocyst inactivation in urothelial cells disrupts autophagy and activates non-canonical NF-κB signaling. In Disease models & mechanisms, 15, . doi:10.1242/dmm.049785. https://pubmed.ncbi.nlm.nih.gov/36004645/
3. Nihalani, Deepak, Solanki, Ashish K, Arif, Ehtesham, Sampson, Matthew G, Lipschutz, Joshua H. 2019. Disruption of the exocyst induces podocyte loss and dysfunction. In The Journal of biological chemistry, 294, 10104-10119. doi:10.1074/jbc.RA119.008362. https://pubmed.ncbi.nlm.nih.gov/31073028/
4. Fujimoto, Brent A, Young, Madison, Carter, Lamar, Fogelgren, Ben, Polgar, Noemi. 2019. The exocyst complex regulates insulin-stimulated glucose uptake of skeletal muscle cells. In American journal of physiology. Endocrinology and metabolism, 317, E957-E972. doi:10.1152/ajpendo.00109.2019. https://pubmed.ncbi.nlm.nih.gov/31593505/
5. Polgar, Noemi, Lee, Amanda J, Lui, Vanessa H, Napoli, Josephine A, Fogelgren, Ben. 2015. The exocyst gene Sec10 regulates renal epithelial monolayer homeostasis and apoptotic sensitivity. In American journal of physiology. Cell physiology, 309, C190-201. doi:10.1152/ajpcell.00011.2015. https://pubmed.ncbi.nlm.nih.gov/26040895/
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