1. Li, Zhipeng, Zhou, Huaxin, Zhai, Xiangyu, Wang, Wei, Jin, Bin. 2023. MELK promotes HCC carcinogenesis through modulating cuproptosis-related gene DLAT-mediated mitochondrial function. In Cell death & disease, 14, 733. doi:10.1038/s41419-023-06264-3. https://pubmed.ncbi.nlm.nih.gov/37949877/
2. Yang, Qinjing, Zeng, Shuangshuang, Liu, Wei. 2023. Roles of cuproptosis-related gene DLAT in various cancers: a bioinformatic analysis and preliminary verification on pro-survival autophagy. In PeerJ, 11, e15019. doi:10.7717/peerj.15019. https://pubmed.ncbi.nlm.nih.gov/36949759/
3. Peng, Yanyu, Shi, Ruimeng, Yang, Siwen, Zhu, Jiayi. . Cuproptosis-related gene DLAT is a biomarker of the prognosis and immune microenvironment of gastric cancer and affects the invasion and migration of cells. In Cancer medicine, 13, e70012. doi:10.1002/cam4.70012. https://pubmed.ncbi.nlm.nih.gov/39031012/
4. Huang, Shuaishuai, Cai, Congbo, Zhou, Kena, Cen, Dong, Weng, Guobin. 2023. Cuproptosis-related gene DLAT serves as a prognostic biomarker for immunotherapy in clear cell renal cell carcinoma: multi-database and experimental verification. In Aging, 15, 12314-12329. doi:10.18632/aging.205181. https://pubmed.ncbi.nlm.nih.gov/37938155/
5. Xu, Lidong, Wu, Peipei, Rong, Aimei, Zhang, Yong, Wu, Huili. 2023. Systematic pan-cancer analysis identifies cuproptosis-related gene DLAT as an immunological and prognostic biomarker. In Aging, 15, 4269-4287. doi:10.18632/aging.204728. https://pubmed.ncbi.nlm.nih.gov/37199628/
智鼠故事 
