1. Chelban, Viorica, Aksnes, Henriette, Maroofian, Reza, Arnesen, Thomas, Houlden, Henry. 2024. Biallelic NAA60 variants with impaired n-terminal acetylation capacity cause autosomal recessive primary familial brain calcifications. In Nature communications, 15, 2269. doi:10.1038/s41467-024-46354-0. https://pubmed.ncbi.nlm.nih.gov/38480682/
2. Donnarumma, Federica, Tucci, Valeria, Ambrosino, Concetta, Altucci, Lucia, Carafa, Vincenzo. 2022. NAA60 (HAT4): the newly discovered bi-functional Golgi member of the acetyltransferase family. In Clinical epigenetics, 14, 182. doi:10.1186/s13148-022-01402-8. https://pubmed.ncbi.nlm.nih.gov/36539894/
3. Gu, Shukai, Li, Junliang, Chen, Weina, Zhang, Cui, Li, Shijie. 2019. ZNF597 is a maternally expressed imprinted gene in the Holstein breed. In Theriogenology, 143, 133-138. doi:10.1016/j.theriogenology.2019.12.007. https://pubmed.ncbi.nlm.nih.gov/31874365/
4. Ahmed, Farjana, Husain, Matloob. 2022. Human N-Alpha-Acetyltransferase 60 Promotes Influenza A Virus Infection by Dampening the Interferon Alpha Signaling. In Frontiers in immunology, 12, 771792. doi:10.3389/fimmu.2021.771792. https://pubmed.ncbi.nlm.nih.gov/35095845/
5. Chen, Xinhui, Shi, Yihua, Fu, Feng, Cen, Zhidong, Luo, Wei. 2024. A Homozygous Variant in NAA60 Is Associated with Primary Familial Brain Calcification. In Movement disorders : official journal of the Movement Disorder Society, 39, 2190-2198. doi:10.1002/mds.30004. https://pubmed.ncbi.nlm.nih.gov/39229657/