Arhgap17 深入了解基因-突变-疾病-模型全维度数据，尽在Rare Disease Data Center（RDDC）综合数据库

MGI:1917747 Mice homozygous for a knock-out allele do not develop spontaneous colitis but show a slight increase in paracellular permeability and increased susceptibility to DSS-induced colitis with rapid destruction of the inner mucus layer.